Key result
Centrally-induced neural vasodilator signals may be transmitted to active skeletal muscle at the onset of voluntary exercise, contributing to exercise hyperemia.
This review proposes that neural vasodilator mechanisms, driven by central command, may contribute to exercise hyperemia at the onset of voluntary exercise in humans, challenging the consensus that sympathetic mechanisms are not involved.
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Does not yet alter exercise prescriptions; leaves open central neural contributions to hyperemia for prospective validation.
Matsukawa et al. (2013) conducted a review in Exercise hyperemia. Centrally-induced neural vasodilator signals may be transmitted to active skeletal muscle at the onset of voluntary exercise, contributing to exercise hyperemia.
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