Highlights the physiological importance of differentiating mechanical block from left ventricular myocardial failure as causes of pulmonary artery hypertension in rheumatic heart disease.
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May aid PAH cause differentiation in rheumatic disease; leaves open prospective validation before practice impact.
Hemodynamic studies were made in 42 patients with rheumatic heart disease with various valvular lesions and in different stages of cardiac function. These studies include observations not only at rest, but also during exercise, following the acute administration of Digoxin, and after recovery from failure. The relationship between symptomatology and hemodynamic findings were considered and the importance of differentiating between mechanical block and left ventricular myocardial failure as causes for pulmonary artery hypertension is stressed.
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FERRER et al. (1952) studied this question.
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