Previous immunocytochemical results from five monkeys with cortical focal epilepsy produced by alumina gel showed a severe decrease at seizure foci of axon terminals that contained glutamic acid decarboxylase (GAD), the synthesizing enzyme for the inhibitory neurotrasmitter, GABA. These data indicated a functional loss of GABAergic terminals but did not show: (1) whether this loss was ' This work was supported
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Ribak et al. (1982) studied this question.