Enterovirus-D68 infection pathogenesis has significant knowledge gaps regarding mechanisms of systemic dissemination, CNS entry, and the induction of acute flaccid myelitis.
Absolute Event Rate: 0% vs 0%
studies. This review highlights the knowledge gaps on the mechanisms of systemic dissemination, routes of entry into the CNS and mechanisms to induce AFM or other CNS complications, as well as the role of virus and host factors in the pathogenesis of EV-D68.
Ayudhya et al. (Thu,) reported a other. Enterovirus-D68 infection pathogenesis has significant knowledge gaps regarding mechanisms of systemic dissemination, CNS entry, and the induction of acute flaccid myelitis.
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