Key result
Chronic heart failure leads to significant cardiopulmonary interactions and pulmonary abnormalities, including reduced lung compliance, impaired diffusion capacity, and sleep-disordered breathing.
Understanding the complex cardiopulmonary interactions in chronic heart failure is essential for optimizing the diagnosis and management of dyspnea and exercise intolerance in these patients.
May aid dyspnea evaluation in chronic HF; leaves open optimal management strategies pending prospective data.
Chronic heart failure places heavy burden on patients, their families and on health care resources, accounting for high numbers of hospital admissions. Despite huge improvements in the treatment of many heart disorders, the clinical syndrome of chronic heart failure as a final pathway of heart pathology is an exception, in that its prevalence is rising, and only small prolongations in survival are occurring. It is associated with high morbidity and poor prognosis, and a survival rate worse than that for some malignant tumors. The reasons for the increasing overall prevalence of chronic heart failure in developed countries lie in prolonged survival owing to modern pharmacological or invasive treatment, better secondary prevention, and aging of the population. Chronic pulmonary disease is common in patients with chronic heart failure. Through sharing some risk factors and overlapping pathophysiological processes, they present diagnostic and therapeutic challenge. The aim of this article is to review various mechanisms responsible for the symptoms of chronic heart failure with consecutive pulmonary interaction and abnormalities in lung function.
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Ivan Alerić (2017) conducted a review in Chronic Heart Failure. Chronic heart failure leads to significant cardiopulmonary interactions and pulmonary abnormalities, including reduced lung compliance, impaired diffusion capacity, and sleep-disordered breathing.
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