What does this research mean for the field?

The hypercoagulable state in cancer patients is driven by a complex interplay of tumor cells directly activating the clotting cascade, inducing procoagulant properties in host cells, and clinical factors such as stasis and iatrogenic complications. Novelty: ClaimNovelty.SYNTHESIS. Consensus alignment: ConsensusAlignment.NEUTRAL.

April 1, 1999Seminars in Thrombosis and Hemostasis

Pathophysiology of the Thrombophilic State in the Cancer Patient

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Population

Cancer patients

Design

Review

Authors

AFAnna FalangaVascular Medicine FRFrederick R. RicklesFlorida State University

Discussion

Member takes

Implication

Supports targeted anticoagulation in cancer; leaves open optimal regimens for prospective trials.

Structured PICO

Population

Cancer patients

Outcome

Pathophysiology of the hypercoagulable state and thrombosis

Understanding the pathophysiology of cancer-associated thrombophilia provides a rationale for more aggressive and specific anticoagulant strategies in selected patients.

Cite This Study

Falanga et al. (1999) studied this question.

synapsesocial.com/papers/6a195d3aa0353395e9583cd1 https://doi.org/10.1055/s-2007-994919

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Pathophysiology of the Thrombophilic State in the Cancer Patient

Discussion

Member takes

Implication

Structured PICO

Cite This Study

Pathophysiology of the Thrombophilic State in the Cancer Patient

Discussion

Member takes

Implication

Structured PICO

Cite This Study

Also Consider

Also Consider