Key result
Severe carbon monoxide poisoning linked to acute reversible cardiomyopathy followed by delayed neuropsychiatric sequelae.
Case Report (n=1)
No
Carbon monoxide poisoning can cause acute cardiomyopathy that may recover quickly, but patients remain at risk for delayed neuropsychiatric sequelae.
Rapid cardiac recovery after CO poisoning does not preclude delayed neuropsychiatric sequelae; leaves open need for prospective monitoring studies.
Carbon monoxide (CO) is an odorless and colorless gas that is formed by the combustion of hydrocarbon. CO poisoning is not an uncommon phenomenon that can have serious consequences for morbidity and mortality. The most frequent causes of accidental poisoning include smoke inhalation from fires, malfunctioning heating devices, smoke from motor vehicles in a poorly ventilated or enclosed space, and electrical cable fires. CO has a higher affinity to hemoglobin than oxygen leading to the formation of carboxyhemoglobin. It impairs the oxygen transport and cytochrome chain which, therefore, leads to further cellular and immunological changes. Here, we present a case of CO poisoning resulting in combined cardiac and neuropsychiatric complications.
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Alarbi et al. (2023) conducted a case report in Carbon monoxide poisoning (n=1). High-flow oxygen and supportive care was evaluated. A 38-year-old woman with severe carbon monoxide poisoning developed acute cardiomyopathy that recovered within a week, followed by delayed neuropsychiatric sequelae one month later.
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