Why the study?
Heart failure pathogenesis may be related to age-dependent decreases in cardiomyocyte respiration, affecting energy-dependent cellular processes.
Older rats with heart failure exhibit significantly reduced cardiomyocyte respiration and altered membrane microviscosity compared to younger rats, suggesting age-dependent cellular energy dysfunction in heart failure.
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Age-related decline in cardiomyocyte respiration in rat HF; leaves open translational relevance to human age-specific mechanisms.
Корепанов et al. (2023) studied this question.
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