Why the study?
To help distinguish stress-induced cardiomyopathy from acute myocardial infarction and acute viral or auto-immune myocarditis, this review summarizes specific mechanisms of myocardial stunning.
This review outlines the pathophysiological mechanisms underlying myocardial stunning in stress-induced cardiomyopathy to aid in distinguishing it from acute myocardial infarction and myocarditis.
Supports imaging differentiation from infarction; leaves open mechanism-targeted interventions pending prospective data.
Stress-induced cardiomyopathy, in contrast to acute myocardial infarction, is a type of acute heart failure characterized by reversible left ventricular dysfunction. Cardiac imaging primarily reveals left ventricle myocardial stunning, 81.7% of which is apical type. Emotional or psychological stress usually precedes the onset of stress-induced cardiomyopathy, which is increasingly being recognized as a unique neurogenic myocardial stunning disease. To distinguish between acute myocardial infarction and acute viral or auto-immune myocarditis, this review summarizes specific mechanisms of myocardial stunning in stress-induced cardiomyopathy, such as calcium disorders, metabolic alterations, anatomical and histological variations in different parts of the left ventricle, and microvascular dysfunction.
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Pei et al. (2022) studied this question.
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