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WITHIN the decade since the syndrome of early infantile autism was first described by Kanner,¹⁻²terms such as childhood schizophrenia,³atypical children,⁴children with unusual sensitivities,⁵and symbiotic psychosis⁶were used to conceptualize similar, yet apparently distinctive clinical entities. The tendency to create separate entities was reinforced by a desire for diagnostic specificity and accuracy and etiologic preference. As the symptomatology in these children varies both with the severity of the illness and age, it has been possible to emphasize distinctive clusters of symptoms and relate these to particular theories of causation. For instance, the predominance of disturbances of relating coupled with the prevailing belief in the 1940's and 1950's that specific syndromes in children must be outgrowths of specific parental behaviors or attitudes₇led to attempts to implicate the parents in the development of early infantile autism. The
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Edward M. Ornitz (1968) studied this question.
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