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January 1, 1999Journal of Biological ChemistryOpen Access

Caspase Cleaved BID Targets Mitochondria and Is Required for Cytochrome c Release, while BCL-XL Prevents This Release but Not Tumor Necrosis Factor-R1/Fas Death

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Authors

AGAtan GrossWeizmann Institute of ScienceXYXiao Ming YinHebei University of EngineeringKWKun WangNational Institute of Biological Sciences, Beijing

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Cite This Study

Gross et al. (1999) studied this question.

synapsesocial.com/papers/6a1d85d533e2df9c962fa643https://doi.org/10.1074/jbc.274.2.1156
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Also Consider

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  1. 1Comparison of the ion channel characteristics of proapoptotic BAX and antiapoptotic BCL-21997 · 493 citations
  2. 2Mutagenesis of the BH3 Domain of BAX Identifies Residues Critical for Dimerization and Killing1998 · 216 citations
  3. 3BH3 Domain of BAD Is Required for Heterodimerization with BCL-XL and Pro-apoptotic Activity1997 · 309 citations