Why the study?
What are the atypical mechanisms of reciprocating tachycardia initiation in patients with Wolff-Parkinson-White syndrome?
What are the atypical mechanisms of reciprocating tachycardia initiation in patients with Wolff-Parkinson-White syndrome?
Identifies atypical mechanisms of reciprocating tachycardia initiation in WPW syndrome, emphasizing the importance of careful electrophysiological assessment for appropriate prophylactic therapy.
Hypothesis-generating in a small WPW case series; leaves open whether these mechanisms alter EP assessment or therapy.
Descriptions of patients with the Wolff-Parkinson-White (WPW) syndrome and reciprocating tachycardia in whom the initiation of the arrhythmia depended neither on the occurrence of premature beats nor on antecedent cycle-length shortening are given. In 5 the occurrence of escape beats in the bundle of His, usually in the presence of sinoatrial disease, activated the tachycardia circuit, but in the other 2 there were unusual mechanisms related to bradycardia-dependent block in the anomalous pathway, and delayed response to shortening of the atrial cycle length, respectively. Careful assessment of such mechanisms is essential for the correct choice of antiarrhythmic prophylactic therapy.
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Coumel et al. (1976) studied this question.
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