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April 1, 1985Biochemical JournalOpen Access

Stimulation of inositol trisphosphate formation in hepatocytes by vasopressin, adrenaline and angiotensin II and its relationship to changes in cytosolic free Ca2+

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Authors

RCR CharestHoward Hughes Medical InstituteVPVeronica PrpićLouisiana State UniversityJEJohn H. ExtonUniversité Paris-Sud

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Charest et al. (1985) studied this question.

synapsesocial.com/papers/6a1f61ce2865985bbe2ac567https://doi.org/10.1042/bj2270079
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Studies on alpha-adrenergic activation of hepatic glucose output. Studies on role of calcium in alpha-adrenergic activation of phosphorylase.1977 · 291 citations
  2. 2Subcellular incorporation of 32P into phosphoinositides and other phospholipids in isolated hepatocytes.1984 · 68 citations
  3. 3Subcellular site and mechanism of vasopressin-stimulated hydrolysis of phosphoinositides in rat hepatocytes.1984 · 88 citations
  4. 4Modification of glycogen synthase activity in isolated rat hepatocytes by tumor-promoting phorbol esters: evidence for differential regulation of glycogen synthase and phosphorylase.1983 · 61 citations
  5. 5Hormone-stimulated metabolism of inositol lipids and its relationship to hepatic receptor function1981 · 207 citations