Key result
Electrophysiologic studies with atrial pacing demonstrated that critical A-V nodal delay was necessary for PSVT induction, supporting A-V nodal reentry without anomalous pathway participation.
Why the study?
What is the mechanism of recurrent PSVT in a patient with type B preexcitation?
Population
1 patient with type B preexcitation and recurrent paroxysmal supraventricular tachycardia (PSVT)
Design
Case_report
Authors
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PSVT in preexcitation may reflect AV nodal reentry; case report leaves open whether pathway ablation prevents recurrence.
Case Report (n=1)
What is the mechanism of recurrent PSVT in a patient with type B preexcitation?
In some patients with preexcitation and PSVT, the tachycardia mechanism may be A-V nodal reentry rather than involving the anomalous pathway, rendering surgical ablation of the pathway ineffective for PSVT prevention.
Kenneth M. Rosen (1973) conducted a case report in Type B preexcitation and recurrent paroxysmal supraventricular tachycardia (PSVT) (n=1). Electrophysiologic studies with atrial pacing was evaluated on Induction of PSVT and mechanism of reentry. Electrophysiologic studies with atrial pacing demonstrated that critical A-V nodal delay was necessary for PSVT induction, supporting A-V nodal reentry without anomalous pathway participation.
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