Answers are grounded in this paper and cite their sources.
Explores estrogen's role in uterine vasodilatation through direct action on smooth muscle cells, indicating significant implications for pregnancy.
To explore the endothelium-independent mechanisms of estrogen induced uterine vasodilatation, this study was performed to determine whether uterine artery smooth muscle (UASM) cells are direct targets of estrogen, and estradiol (E2) stimulates extracellular signal-regulated kinases (ERK2/1) in endothelium denuded UASMs.
No takes yet. Share an insight, caveat, or question.
Lee et al. (2011) studied this question.
Synapse has enriched 10 related papers. Consider them for comparative context: