Successive viral infections exacerbate myocardial damage in murine models, and antimyosin antibodies target degenerating myocytes, highlighting mechanisms of progression from viral myocarditis to cardiomyopathy.
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May prompt vigilance for recurrent viral exposures in myocarditis; hypothesis-generating in animal models, leaves open human relevance.
Matsumori et al. (1991) studied this question.
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