Key result
High trait anger in adolescents is linked to greater cardiovascular reactivity during anger provocation.
Why the study?
Does high trait anger increase cardiovascular reactivity to anger provocation in adolescents?
Cross-Sectional (n=201)
No
Does high trait anger increase cardiovascular reactivity to anger provocation in adolescents?
The study demonstrates that Spielberger's state-trait theory of anger applies to adolescents, with high trait anger linked to increased cardiovascular reactivity and psychosocial impairment.
Answers are grounded in this paper and cite their sources.
High trait anger linked to greater CV reactivity in adolescents; extends adult theory but leaves prospective validation open.
Spielberger's state-trait theory of anger was investigated in adolescents (n = 201, ages 10-18, 53% African American, 47% European American, 48% female) using Deffenbacher's five hypotheses formulated to test the theory in adults. Self-reported experience, heart rate (HR), systolic blood pressure (SBP), and diastolic blood pressure (DBP) responses to anger provoking imagery scripts found strong support for the application of this theory to adolescents. Compared with the low trait anger (LTA) group, adolescents with high trait anger (HTA) produced increased HR, SBP, and DBP, and greater self-report of anger to anger imagery (intensity hypothesis) but not greater self-report or cardiovascular reactivity to fear or joy imagery (discrimination hypothesis). The HTA group also reported greater frequency and duration of anger episodes and had longer recovery of SBP response to anger (elicitation hypothesis). The HTA group was more likely to report negative health, social, and academic outcomes (consequence hypothesis). Adolescents with high hostility reported more maladaptive coping with anger, with higher anger-in and anger-out than adolescents with low hostility (negative expression hypothesis). The data on all five hypotheses supported the notion that trait anger is firmly entrenched by the period of adolescence, with few developmental differences noted from the adult literature.
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Quinn et al. (2013) conducted a cross-sectional in High trait anger (n=201). High trait anger vs. Low trait anger was evaluated on Cardiovascular reactivity (composite of HR, SBP, and DBP) to anger provocation imagery (p=<0.05). Adolescents with high trait anger demonstrated greater cardiovascular reactivity (HR, SBP, DBP) and self-reported anger to anger-provoking imagery compared to those with low trait anger (p<0.05).
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