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Editorial
This is a commentary discussing the role of the 'dead-end tract' as a substrate for arrhythmogenesis.
In the review, now published as part of the special issue devoted to the development of the conduction tissues, de Vries and his colleagues discuss the potential role of the so-called "dead-end tract" as a substrate for arrhythmogenesis [1].[...].
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Anderson et al. (2016) studied this question.
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