Key result
Cardiac hypertrophy secondary to swimming exercise resulted in an increased density of adrenergic structures, indicating newly formed myocardial blood vessels acquire a normal adrenergic nerve supply.
Why the study?
Does the method of inducing experimental cardiac hypertrophy affect the sympathetic myocardial innervation in rats?
Does the method of inducing experimental cardiac hypertrophy affect the sympathetic myocardial innervation in rats?
Experimental cardiac hypertrophy induced by swimming exercise, but not aortic stenosis, is associated with newly formed myocardial blood vessels acquiring a normal adrenergic nerve supply.
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Findings suggest model-dependent sympathetic reinnervation in rat hypertrophy; leaves open applicability to human disease.
Unge et al. (1973) studied Experimental cardiac hypertrophy. Renal hypertension, aortic stenosis, and swimming exercise was evaluated on Density of adrenergic structures and sympathetic innervation of the hypertrophied myocardium. Cardiac hypertrophy secondary to swimming exercise resulted in an increased density of adrenergic structures, indicating newly formed myocardial blood vessels acquire a normal adrenergic nerve supply.
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