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March 12, 2003Proceedings of the National Academy of SciencesOpen Access

A gain-of-function mutation in ftsA bypasses the requirement for the essential cell division gene zipA in Escherichia coli

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Authors

BGBrett GeisslerEcolab (United States)DEDany ElrahebThe University of Texas Health Science Center at HoustonWMWilliam MargolinThe University of Texas Health Science Center at Houston

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Geissler et al. (2003) studied this question.

synapsesocial.com/papers/6a26e1bb62c511ceafb39b26https://doi.org/10.1073/pnas.0635003100
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Role of the Carboxy Terminus of <i>Escherichia coli</i> FtsA in Self-Interaction and Cell Division2000 · 62 citations
  2. 2Dominant C‐terminal deletions of FtsZ that affect its ability to localize in <i>Caulobacter</i> and its interaction with FtsA1998 · 121 citations
  3. 3Escherichia coli Division Inhibitor MinCD Blocks Septation by Preventing Z-Ring Formation2001 · 121 citations
  4. 4A widely conserved bacterial cell division protein that promotes assembly of the tubulin-like protein FtsZ2002 · 402 citations
  5. 5ZipA Is Required for Recruitment of FtsK, FtsQ, FtsL, and FtsN to the Septal Ring in <i>Escherichia coli</i>2002 · 106 citations