SFL-VIR-03 Viral Cross-Species Transmission Represents Latent G₀ Architecture Expression Rather Than Evolutionary Leap

Cross-species infection does not originate from newly evolved viral capabilities. Instead, it refers to the environmental activation of latent adaptive traits that are preset within the original G₀ framework. All genomic changes occurring during transmission only produce cumulative genetic damage ΔG_d and information loss, which cannot reconstruct core viral structures or create brand-new cross-species adaptation mechanisms. Combined with the unified system state equation: G=G₀+ΔG_d(+)-ΔG_d(-) In accordance with the universal dissipation stability constraint of complex systems: ε(T)=o(1/T) This paper verifies that time accumulation and repeated replication cannot break the fixed species boundary defined by G₀ architecture. All cross-host spillover phenomena belong to intrasystem latent trait expression within the fixed framework, rather than interspecific or cross-kingdom evolutionary jumps. This interpretation eliminates multiple logical inconsistencies in conventional spillover theories. Furthermore, it achieves perfect isomorphic correspondence with the latent reserve mechanism and fault tolerance structure of biological cells and electronic hardware systems recorded in the SFL-EAI serial papers, further improving the universality of the SFL unified complex system theory.