Key result
Overexpression of HSPA12B attenuated cardiac dysfunction and remodelling at 1 and 4 weeks after myocardial infarction in mice through an eNOS-dependent mechanism.
Why the study?
Does HSPA12B overexpression improve cardiac dysfunction and remodeling after myocardial infarction in mice?
Does HSPA12B overexpression improve cardiac dysfunction and remodeling after myocardial infarction in mice?
No takes yet. Share an insight, caveat, or question.
Identifies HSPA12B as a potential therapeutic target for post-MI remodeling; leaves.
Li et al. (2013) studied Myocardial infarction. Overexpression of HSPA12B vs. Wild-type (WT) littermates was evaluated on Cardiac dysfunction and remodelling. Overexpression of HSPA12B attenuated cardiac dysfunction and remodelling at 1 and 4 weeks after myocardial infarction in mice through an eNOS-dependent mechanism.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: