Key result
Aortic blood acceleration increases ~3.5-fold from the LVOT to the sinotubular junction.
This physiological study proposes that electromagnetic interactions contribute to arterial blood flow, driven by observed acceleration increases that contradict classical fluid dynamics.
Does not support changes in clinical practice; hypothesis-generating for electromagnetic contributions to arterial flow.
Blood flow acceleration increases from the left ventricular outflow tract, to the sinotubular junction and the ascending aorta, while it must be decreasing due to the flow turbulences in the Valsalva sinuses and increased diameter of the vessel. Total energy of the pulse wave in the arterioles is up to 7.2 times higher, than in the ascending aorta, while it must be low due to the energy dissipation in the viscous flow, with the distance from the heart. Purpose of the study is identifying the additional possible energy source, for the arterial blood flow.
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Beraia et al. (2018) studied Healthy (n=12). Echocardiography and ECG-gated MRI was evaluated on Blood acceleration ratio from the left ventricular outflow tract to the sinotubular junction (assessed via MRI) (3.5 ± 0.3 times higher). Blood acceleration from the left ventricular outflow tract to the sinotubular junction was 3.5 times higher, leading the authors to hypothesize that electromagnetic forces contribute to arterial blood flow.
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