Key result
A systemic immune-inflammation index cut-off of 1,228,000 predicted new-onset atrial fibrillation following STEMI with 60% sensitivity and 78.1% specificity.
Why the study?
The predictive capacity of the systemic immune-inflammation index for detecting new-onset atrial fibrillation following STEMI had not been established.
Does the systemic immune-inflammation index predict new-onset atrial fibrillation following STEMI?
Observational (n=402)
Does the systemic immune-inflammation index predict new-onset atrial fibrillation following STEMI?
Answers are grounded in this paper and cite their sources.
The systemic immune-inflammation index is an independent predictor of new-onset atrial fibrillation after STEMI, performing similarly to hs-CRP and better than NLR or PLR.
Aim: To investigate the predictive capacity of a systemic immune–inflammation index (SII) in detecting new-onset atrial fibrillation (NOAF) following ST segment elevation myocardial infarction (STEMI). Patients & methods: A total of 402 STEMI patients were enrolled in the study. The patients were divided into two groups according to NOAF development. Results: A cut-off point of 1,228,000 for SII was identified with 60% sensitivity and 78.1% specificity to predict NOAF following STEMI. According to pairwise analysis of receiver operating characteristic curve analysis, the predictive power of SII in detecting NOAF following STEMI was similar to high-sensitive C-reactive protein, and better than neutrophil-to-lymphocyte ratio or platelet-to-lymphocyte ratio. Conclusion: SII can be used as one of the independent predictors of NOAF following STEMI.
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Bağcı et al. (2021) conducted an observational in ST segment elevation myocardial infarction (STEMI) (n=402). Systemic immune-inflammation index (SII) was evaluated on New-onset atrial fibrillation (NOAF). A systemic immune-inflammation index cut-off of 1,228,000 predicted new-onset atrial fibrillation following STEMI with 60% sensitivity and 78.1% specificity.
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