Key result
Caffeine's arrhythmogenic and electrophysiologic effects were evaluated in 19 participants (7 normal volunteers and 12 patients with heart disease), but results are truncated in the abstract.
Observational (n=19)
Caffeine restriction may not reduce arrhythmia risk; leaves open questions on long-term effects in larger cohorts.
IT has been estimated that in a typical year Americans consume nearly 109 kg of coffee.1 In addition, large amounts of tea, cola, and other caffeinated substances are ingested.Caffeine has long been implicated in the causation of arrhythmias, but studies in human beings have been limited. This study was undertaken to determine the cardiac electrophysiologic effects of caffeine, as well as caffeine's arrhythmogenic potential, in normal volunteers and patients with heart disease.MethodsSeven normal volunteers with a mean age of 24 years (range, 20 to 31) and 12 patients with a mean age of 43 years (range, 17 . . .
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Dobmeyer et al. (1983) conducted an observational in Heart disease and normal volunteers (n=19). Caffeine was evaluated on Cardiac electrophysiologic effects and arrhythmogenic potential. Caffeine's arrhythmogenic and electrophysiologic effects were evaluated in 19 participants (7 normal volunteers and 12 patients with heart disease), but results are truncated in the abstract.
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