This state-of-the-art review examines the mechanical performance of ultrathin-strut drug-eluting stents using finite element analysis, focusing on radial expansion and stent-vessel stresses.
What is the mechanical performance of ultrathin-strut drug-eluting stents evaluated via finite element analysis?
This state-of-the-art review highlights the utility of finite element analysis in evaluating the mechanical performance and stresses of ultrathin-strut drug-eluting stents during PCI.
The latest drug-eluting stents (DESs) are the gold standard for patient treatment during percutaneous coronary intervention (PCI). The latest advancements in DES innovation have resulted in the development of new stent technologies with reduced thickness in the struts. The new DES design, ultrathin-strut DESs, features struts measuring less than 70 μm in thickness. The evidence for these devices is derived from observational studies, extensive meta-analyses, and randomized trials with long-term outcomes. The investigation is focused on determining the comparative performance of ultrathin-strut DESs and conventional new-generation DESs across various clinical settings and patient lesion profiles. The objective of the seminar is to examine recent advancements in the use of very thin DESs and the potential of computational modeling in coronary arteries during PCI. An analysis of the mechanical performance of ultrathin DESs has been conducted in terms of radial expansion and stresses within the stent-vessel system. Residual stresses generated by the crimping process will also be considered.
Nappi et al. (Wed,) conducted a review in Percutaneous coronary intervention. Ultrathin-strut drug-eluting stents and finite element analysis vs. Conventional new-generation drug-eluting stents was evaluated on Mechanical performance in terms of radial expansion and stresses within the stent-vessel system. This state-of-the-art review examines the mechanical performance of ultrathin-strut drug-eluting stents using finite element analysis, focusing on radial expansion and stent-vessel stresses.