Key result
Therapeutic strategies targeting integrin-mediated adhesion and chemokine receptors are proposed to control T. cruzi-elicited unbalanced inflammation in Chagas disease.
Population
Murine models for Chagas disease and patients with Chagas disease
Design
Review
Authors
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Mechanisms of chagasic myocarditis progression remain unclear; leaves open the roles of parasite persistence versus autoimmunity.
This review proposes targeting integrin-mediated adhesion and chemokine receptors as a potential therapeutic strategy to control chronic inflammation in Chagas disease myocarditis.
Joseli Lannes-Vieira (2003) conducted a review in Chagas disease. Therapeutic strategies targeting integrin-mediated adhesion and chemokine receptors was evaluated. Therapeutic strategies targeting integrin-mediated adhesion and chemokine receptors are proposed to control T. cruzi-elicited unbalanced inflammation in Chagas disease.
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