Key result
Short-term alcohol ingestion by normotensive 'social' drinkers did not increase office or ambulatory blood pressure compared to an isocaloric sucrose substitute or no intervention.
Why the study?
Does short-term alcohol consumption affect office and ambulatory blood pressure in normotensive 'social' drinkers?
Does short-term alcohol consumption affect office and ambulatory blood pressure in normotensive 'social' drinkers?
Short-term alcohol ingestion by normotensive 'social' drinkers does not increase office or ambulatory blood pressure.
Supports no acute BP rise from social alcohol in normotensives; leaves open chronic effects and hypertensive populations.
Office blood pressure (OBP), ambulatory blood pressure (ABP), and vascular reactivity were measured in 12 normotensive 'social' drinkers (mean alcohol consumption of 9.7 +/- 3.1 g/day) after 4 days of alcohol consumption (1 g/kg/day) and compared with the effects of an isocaloric substitute (sucrose, 1.75 g/kg/day) or no intervention. Alcohol consumption did not affect OBP or 24-mean ABP but appeared to decrease nocturnal systolic and diastolic blood pressure soon after consumption. Although vascular reactivity did not differ between the groups, the blood pressure rise in response to isometric exercise tended to be lower following alcohol. Thus, short-term alcohol ingestion by normotensive 'social' drinkers does not increase office or ambulatory blood pressure.
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O’Callaghan et al. (1995) studied Normotensive 'social' drinkers (n=12). Alcohol consumption vs. Isocaloric substitute (sucrose, 1.75 g/kg/day) or no intervention was evaluated on Office blood pressure (OBP) and ambulatory blood pressure (ABP). Short-term alcohol ingestion by normotensive 'social' drinkers did not increase office or ambulatory blood pressure compared to an isocaloric sucrose substitute or no intervention.
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