Key result
Short-term exposure to environmentally relevant concentrations of secondhand smoke significantly reduced heart rate variability and increased ventricular arrhythmia susceptibility in mice.
Why the study?
Does short-term secondhand smoke exposure decrease heart rate variability and increase arrhythmia susceptibility in mice?
Does short-term secondhand smoke exposure decrease heart rate variability and increase arrhythmia susceptibility in mice?
Short-term exposure to secondhand smoke at environmentally relevant concentrations decreases heart rate variability and increases susceptibility to ventricular arrhythmias and AV block in a mouse model.
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Alerts to potential proarrhythmic effects of secondhand smoke; hypothesis-generating in mice and requires human validation.
Chen et al. (2008) studied this question. Short-term secondhand smoke (SHS) exposure vs. Filtered air was evaluated on Heart rate variability (HRV) and susceptibility to arrhythmia. Short-term exposure to environmentally relevant concentrations of secondhand smoke significantly reduced heart rate variability and increased ventricular arrhythmia susceptibility in mice.
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