If the epidermis of an animal is injured so that its continuity is destroyed, the surrounding cells multiply and spread over the wound until the gap is filled. Then their growth ceases. The factors which initiate this renewed growth of “repair”, and the factors by which growth is arrested when the defect has been made good, are very incompletely known. The object of the present work was to re-investigate this old problem upon new material.
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V. B. Wigglesworth (1937) studied this question.
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