Reveals fluoride exposure disrupts calcium signaling and mitochondrial function in enamel cells, suggesting a mechanism for dental fluorosis.
refilling of the ER. Fluoride negatively affected mitochondrial respiration, elicited mitochondrial membrane depolarization, and disrupted mitochondrial morphology. Together, these data provide a potential mechanism underlying dental fluorosis.
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Aulestia et al. (2020) studied this question.
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