β-Amyloid Induces Paired Helical Filament-like Tau Filaments in Tissue Culture

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Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1. 1Sequential phosphorylation of Tau by glycogen synthase kinase‐3β and protein kinase A at Thr212 and Ser214 generates the Alzheimer‐specific epitope of antibody AT100 and requires a paired‐helical‐filament‐like conformation1998 · 325 citations
2. 2Protofibrillar intermediates of amyloid beta-protein induce acute electrophysiological changes and progressive neurotoxicity in cortical neurons.1999 · 921 citations
3. 3Tau is a candidate gene for chromosome 17 frontotemporal dementia1998 · 1,391 citations
4. 4Protofibrillar Intermediates of Amyloid β-Protein Induce Acute Electrophysiological Changes and Progressive Neurotoxicity in Cortical Neurons1999 · 817 citations
5. 5Straight and paired helical filaments in Alzheimer disease have a common structural unit.1991 · 374 citations