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September 1, 1982Journal of Clinical InvestigationOpen Access

Ability of the xid gene to prevent autoimmunity in (NZB X NZW)F1 mice during the course of their natural history, after polyclonal stimulation, or following immunization with DNA.

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Authors

BSBernhard SteinbergPSP A SmathersKFKatrine Hartmund Frederiksen

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Cite This Study

Steinberg et al. (1982) studied this question.

synapsesocial.com/papers/6a6f46638031ec7bb1dbade0https://doi.org/10.1172/jci110651
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Also Consider

Synapse has enriched 4 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Role of accessory cells in B cell activation. II. The interaction of B cells with accessory cells results in the exclusive activation of an Lyb5+ B cell subpopulation.1980 · 54 citations
  2. 2Effect of CBA/N xid on Spontaneous Production of Antibodies to DNA in MRL/1 and NZB Backcross Mice1980 · 12 citations
  3. 3CBA/N X-linked B-cell defect prevents NZB B-cell hyperactivity in F1 mice.1979 · 72 citations
  4. 4GENETIC CONTROL OF THE ANTIBODY RESPONSE TO TYPE III PNEUMOCOCCAL POLYSACCHARIDE IN MICE1972 · 344 citations