Why the study?
Does hyperbaric oxygen exposure alter vascular NO bioavailability and eNOS activity in rat thoracic aorta?
Does hyperbaric oxygen exposure alter vascular NO bioavailability and eNOS activity in rat thoracic aorta?
Hyperbaric oxygen reduces endothelium-derived NO bioavailability and alters vascular reactivity through extraendothelial NO production, independent of eNOS catalytic activity reduction.
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HBO may impair endothelium-dependent relaxation via novel mechanisms; hypothesis-generating in animal models and leaves open in vivo or clinical translation.
Hink et al. (2006) studied this question.
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