Population
HL-1 cardiac cells subjected to serum- and amino acid-free conditions for 24h
Comparison
UA-8 (13-tridec-8-enoic acid, 1μM) or 14,15-EET vs Untreated starved HL-1 cells and non-starved…
Design
Preclinical, blind morphometric analysis
Follow-up
24 hours
Authors
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EETs preserve mitochondrial quality in stressed cardiomyocytes; leaves open whether this extends to in vivo cardioprotection.
EET-mediated events preserve mitochondrial function and quality in cardiac cells during starvation-induced stress.
El‐Sikhry et al. (2016) studied this question.
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