Population
Human embryonic kidney cells transiently transfected with wild-type or mutant cDNA alone or in combination…
Comparison
Expression of mutant D1790G SCN5A cDNA vs Expression of wild-type hH1 SCN5A cDNA
Design
Preclinical
Authors
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May alter LQT3 mechanism assumptions in models; leaves open whether subunit interactions affect human risk stratification or therapy.
The novel LQT-3 mutation D1790G alters sodium channel inactivation through interactions between alpha- and beta1-subunits, rather than causing sustained inward current typical of other LQT-3 mutations.
An et al. (1998) studied this question.
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