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September 1, 1986Journal of Biological ChemistryOpen Access

Ca2+ regulates the inositol tris/tetrakisphosphate pathway in intact and broken preparations of insulin-secreting RINm5F cells.

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Authors

TBTrevor J. BidenGarvan Institute of Medical ResearchCWClaes B. WollheimLinköping University

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Biden et al. (1986) studied this question.

synapsesocial.com/papers/6a6fdd6375498292b7093ddbhttps://doi.org/10.1016/s0021-9258(18)67180-7
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Inositol 1,4,5-trisphosphate and inositol 1,3,4-trisphosphate formation in Ca2+-mobilizing-hormone-activated cells1985 · 236 citations
  2. 2Rapid formation of inositol 1,3,4,5-tetrakisphosphate following muscarinic receptor stimulation of rat cerebral cortical slices1985 · 646 citations
  3. 3Short Communication Inositol Tetrakis- And Pentakisphosphates In Gh4 Cells1985 · 202 citations
  4. 4Metabolism of inositol 1,4,5-trisphosphate and inositol 1,3,4-trisphosphate in rat parotid glands1985 · 453 citations
  5. 5Inositol 1,4,5-trisphosphate mobilizes intracellular Ca2+ from permeabilized insulin-secreting cells1984 · 184 citations