Why the study?
Do N-acetylcysteine or diphenyleneiodonium reduce TGF-beta1-induced fibronectin secretion and alpha-SMA expression in human lung fibroblasts?
Do N-acetylcysteine or diphenyleneiodonium reduce TGF-beta1-induced fibronectin secretion and alpha-SMA expression in human lung fibroblasts?
NADPH oxidase-dependent ROS play a critical role in TGF-beta1-induced fibronectin secretion and alpha-SMA expression in human lung fibroblasts, highlighting a potential mechanism in progressive lung fibrosis.
ROS inhibition attenuates fibroblast activation in vitro; hypothesis-generating for antifibrotic strategies in lung fibrosis.
No abstract yet. The journal hasn't published one we can show.
No takes yet. Share an insight, caveat, or question.
Ha et al. (2005) studied this question.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: