Population
Smooth muscle
Design
Review
Authors
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Does not alter vasomotor therapies; leaves open whether phospholipase C or myosin phosphorylation pathways are viable targets.
Flash photolysis of caged compounds reveals that the delay in smooth muscle contraction following agonist stimulation is primarily driven by phospholipase C activation and InsP3 production, with force development limited by myosin light chain phosphorylation.
Somlyo et al. (1990) studied this question.
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