Escherichia coli mutants defective in the fip gene contain reduced levels of thioredoxin activity. F' derivatives of trxA mutants failed to support f1 growth, although suppressors of an amber trxA mutation restored both thioredoxin activity and the Fip+ phenotype. Plasmids carrying the trxA gene restored the Fip+ phenotype to fip strains.
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Lim et al. (1985) studied this question.
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