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December 1, 1999Critical Care Medicine

Differential expression pattern of heme oxygenase-1/heat shock protein 32 and nitric oxide synthase-II and their impact on liver injury in a rat model of hemorrhage and resuscitation

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Authors

HRHauke RensingUniversity of FreiburgIBInge BauerDüsseldorf University HospitalVDVerena Datene

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Rensing et al. (1999) studied this question.

synapsesocial.com/papers/6a70bd3626a7f98052dd33d5https://doi.org/10.1097/00003246-199912000-00027
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Also Consider

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  1. 1Activation of soluble guanylate cyclase by carbon monoxide and inhibition by superoxide anion1990 · 183 citations
  2. 2Purification and properties of bovine spleen heme oxygenase. Amino acid composition and sites of action of inhibitors of heme oxidation.1982 · 155 citations
  3. 3INHIBITION OF NITRIC OXIDE SYNTHESIS DURING SEVERE SHOCK BUT NOT AFTER RESUSCITATION INCREASES HEPATIC INJURY AND NEUTROPHIL ACCUMULATION IN HEMORRHAGED RATS1995 · 55 citations
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  5. 5Hemorrhage induces enhanced Kupffer cell cytotoxicity while decreasing peritoneal or splenic macrophage capacity. Involvement of cell-associated tumor necrosis factor and reactive nitrogen1991 · 78 citations