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Mechanistic analysis uncovers how defective viruses stimulate resting CD4 T lymphocytes, suggesting a hidden driver of persistent infection and pathogenesis.
Many aspects of the human immunodeficiency virus (HIV) life cycle are well understood, yet a key paradox remains. The primary targets for HIV are resting CD4 T lymphocytes that are not permissive to HIV replication unless activated by some external and independent event. Numerous mechanisms for this
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Finzi et al. (2006) studied this question.
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