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September 1, 2000Journal of Biological ChemistryOpen Access

Participation of Smad2, Smad3, and Smad4 in Transforming Growth Factor β (TGF-β)-induced Activation of Smad7

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Authors

MSMarcin StopaPoznan University of Medical SciencesDADirk AnhufRWTH Aachen UniversityLTLara TerstegenBeiersdorf (Germany)

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Stopa et al. (2000) studied this question.

synapsesocial.com/papers/6a70e4c326a7f98052dd598fhttps://doi.org/10.1074/jbc.m003282200
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Smad3-Smad4 and AP-1 Complexes Synergize in Transcriptional Activation of the c-Jun Promoter by Transforming Growth Factor β1999 · 262 citations
  2. 2Smad6 inhibits BMP/Smad1 signaling by specifically competing with the Smad4 tumor suppressor1998 · 724 citations
  3. 3Characterization of Human FAST-1, a TGFβ and Activin Signal Transducer1998 · 230 citations
  4. 4Smad4/DPC4 and Smad3 Mediate Transforming Growth Factor-β (TGF-β) Signaling through Direct Binding to a Novel TGF-β-responsive Element in the Human Plasminogen Activator Inhibitor-1 Promoter1998 · 139 citations
  5. 5The tumor suppressor Smad4/DPC4 and transcriptional adaptor CBP/p300 are coactivators for Smad3 in TGF-β-induced transcriptional activation1998 · 531 citations