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June 15, 1998Development

Mice lacking the homeodomain transcription factor Nkx2.2 have diabetes due to arrested differentiation of pancreatic β cells

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Authors

LSLori SusselUniversity of Colorado SystemJKJulie KalamarasUniversity of California, San FranciscoDHDennis J. Hartigan-O’ConnorUniversity of California, San Francisco

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Cite This Study

Sussel et al. (1998) studied this question.

synapsesocial.com/papers/6a70f1c7f44fa9f079deb16fhttps://doi.org/10.1242/dev.125.12.2213
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Also Consider

Synapse has enriched 4 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Long-Term Course of Neonatal Diabetes1995 · 239 citations
  2. 2Developmental expression of the prohormone convertases PC1 and PC2 in mouse pancreatic islets1994 · 26 citations
  3. 3Glucokinase as Glucose Sensor and Metabolic Signal Generator in Pancreatic β-Cells and Hepatocytes1990 · 624 citations
  4. 4Diabetes, defective pancreatic morphogenesis, and abnormal enteroendocrine differentiation in BETA2/NeuroD-deficient mice1997 · 1,033 citations