Demonstrates the feasibility of performing ablation for cavotricuspid isthmus-dependent flutter in the presence of a mechanical tricuspid valve.
Case report details CTI arrhythmogenic anatomy; leaves open whether input-specific mapping refines flutter ablation.
The cavotricuspid isthmus is an exceptionally arrhythmogenic structure due to a combination of particular anatomical and electrophysiological characteristics. It is a relatively narrow corridor of myocardium, bounded by anatomical conduction block at the ventricular aspect by the tricuspid annulus, and atrially by electrically inert portions of the inferior vena cava. Electrical wavefronts may propagate into the CTI via septal and lateral inputs, that are characterized by myocyte anisotropy and relatively slow conduction velocity.
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Padfield et al. (2024) studied this question.
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