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March 1, 1994Molecular and Cellular BiologyOpen Access

Functional and physical interaction between p53 and BZLF1: implications for Epstein-Barr virus latency.

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Authors

QZQ ZhangTongji UniversityDGDavid GutschMerck & Co., Inc., Rahway, NJ, USA (United States)SKShannon C. KenneyUniversity of Wisconsin–Madison

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Cite This Study

Zhang et al. (1994) studied this question.

synapsesocial.com/papers/6a714ea18031ec7bb1dce260https://doi.org/10.1128/mcb.14.3.1929
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Also Consider

Synapse has enriched 4 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1The cellular oncogene c-myb can interact synergistically with the Epstein-Barr virus BZLF1 transactivator in lymphoid cells.1992 · 48 citations
  2. 2Expression of Epstein–Barr virus replicative proteins in aids‐related non‐Hodgkin's lymphoma cells1991 · 114 citations
  3. 3An enhancer within the divergent promoter of Epstein-Barr virus responds synergistically to the R and Z transactivators1990 · 152 citations
  4. 4In vitro transcriptional activation, dimerization, and DNA-binding specificity of the Epstein-Barr virus Zta protein1990 · 137 citations