Why the study?
How coronary microvascular resistance is coupled with myocardial oxygen consumption remains contested, confounded by debates on experimental outcomes, circular interpretations of coronary venous Po 2, and unaccounted-for influences.
Design
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Mechanisms of coronary flow-metabolic coupling remain debated; leaves open targeted mechanistic research without changing clinical practice.
Key points are not available for this paper at this time.
Tune et al. (2019) studied this question.
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