Shows increased apoptosis and DNA damage in DLBCL cells using dual inhibitors, indicating a promising treatment strategy.
values of 19.3 nM in U2932 cells and 9.1 nM in KARPAS-422 cells. Compared to single-target inhibitors, its dual inhibitory action significantly increased the apoptosis rate and induced substantial DNA damage in DLBCL cells.
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Han et al. (2026) studied this question.
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