Why the study?
Cells can misidentify transposable elements as viruses and trigger type I interferon responses, making a better biological understanding of transposable element replication and antiviral nucleic acid sensing essential for treating associated diseases.
Design
Review
Authors
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TEs' co-option for antiviral regulation and embryogenesis extends evolutionary insights; leaves open mechanistic roles in human disease.
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Gázquez-Gutiérrez et al. (2021) studied this question.
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