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May 1, 1999Cardiovascular ResearchOpen Access

Structure and function of the cardiac sodium channels

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JBJeffrey R. Balser

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Overview

Structural insights into Na channels do not alter current arrhythmia management; leaves open targeted drug discovery pending clinical validation.

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Cite This Study

Jeffrey R. Balser (1999) studied this question.

synapsesocial.com/papers/6a71a0c7439bab0cabc3a23ehttps://doi.org/10.1016/s0008-6363(99)00031-0
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Molecular Localization of an Ion-Binding Site Within the Pore of Mammalian Sodium Channels1992 · 278 citations
  2. 2Phenotypic Characterization of a Novel Long-QT Syndrome Mutation (R1623Q) in the Cardiac Sodium Channel1998 · 142 citations
  3. 3Assignment of the human heart tetrodotoxin-resistant voltage-gated Na <sup>+</sup> channel α-subunit gene (SCN5A) to band 3p212008 · 125 citations
  4. 4Differences in steady-state inactivation between Na channel isoforms affect local anesthetic binding affinity1997 · 78 citations
  5. 5Isoproterenol, DBcAMP, and forskolin inhibit cardiac sodium current1989 · 126 citations